Do you have any idea how poisonous CO2 is? Carbon dioxide is the poison you breath out when you exhale. It is real. It is not fake. You learn about carbon dioxide when you are a child in kindergarten. You learn about the life cycle of a plant. Carbon dioxide is in the air, but exhaled because it is poison and can’t be used, but plants use that element to create oxygen for us – our life source.
So if we exhale poisonous co2 every time we breathe, no wonder I feel anxious when I put on a mask.
Let’s ask questions:
Is a mask a barrier?
Why wear a mask if it is not a barrier?
How does it prevent virus?
If a mask is a barrier, how does it allow circulation?
If a mask is a barrier, how does poisonous CO2 circulate?
If a mask is a barrier, and poisonous CO2 does not circulate, how is that healthy?
How is it okay for a barrier, which allows poisonous CO2 to be trapped and renter my lungs recommended for 7 hours on a child?
If the mask is circulating properly, how is it preventing a virus?
If a mask is circulating and CO2 is a non-issue, then it is NOT PREVENTING A VIRUS.
A mask is then deemed unnecessary, and must not be a mandate.
No wonder I couldn’t think properly when I would shop with a mask on. I was being poisoned by my own breath. I had terrible headaches, felt exhausted all the time. Masks are nothing but trouble. It’s proven they don’t prevent virus from entering our systems. You can get a virus from the liquid in your eyes. It can enter through your ears…
Masks are stupid. You are poisoning your children. Yet, this is America, the land of the free. So if you want to keep wearing them, please do. I choose not to. I want that choice
What are the risks involved in wearing masks? Why don’t we just comply like everyone else?
- Over exposure to CO2 -The study included 11 healthy volunteers, median age 32 years (range 16-54) and 6 (55%) men. Percent mean (SD) changes in CO2 values for no mask, JustAir® PAPR, KN95 respirator and valve respirator were 0.26 (0.12), 0.59 (0.097), 2.6 (0.14) and 2.4 (0.59), respectively. Use of face masks (KN95 and valved-respirator) resulted in significant increases in CO2 concentrations, which exceeded the 8-h NIOSH exposure threshold limit value-weighted average (TLV-TWA). However, the increases in CO2 concentrations did not breach short-term (15-min) limits. Importantly, these levels were considerably lower than the long-term (8-h) NIOSH limits during donning JustAir® PAPR. There was a statistically significant difference between all pairs (p < 0.0001, except KN95 and valved-respirator (p = 0.25).
Conclusion: Although, significant increase in CO2 concentrations are noted with routinely used face-masks, the levels still remain within the NIOSH limits for short-term use. Therefore, there should not be a concern in their regular day-to-day use for healthcare providers. The clinical implications of elevated CO2 levels with long-term use of face masks needs further studies. Use of PAPR prevents relative hypercapnoea.
What happens during hypercapnia?
Hypoxia can also lead to a condition called hypercapnia. This occurs when the lungs retain too much carbon dioxide due to breathing difficulties. When you can’t breathe in, it’s likely you won’t be able to breathe out as you should. This may elevate your carbon dioxide levels in your bloodstream, which can be DEADLY
What Happens When You Have Hypercapnia?
Hypercapnia changes the pH balance of your blood, making it too acidic. This can happen slowly or suddenly. If it happens slowly, your body may be able to keep up by making your kidneys work harder. Your kidneys release and reabsorb bicarbonate, a form of carbon dioxide, which helps keep your body’s pH level balanced.
A sudden rise in carbon dioxide, called acute hypercapnia, is more dangerous, because your kidneys can’t handle the spike. This is most likely to happen if you have a severe case of COPD or if you get a flare-up. Either way, you may be breathing too slowly, which means you aren’t taking in air and letting out carbon dioxide at a healthy rate
Hypercapnia, or hypercarbia, is a condition that arises from having too much carbon dioxide in the blood.
It is often caused by hypoventilation or disordered breathing where not enough oxygen enters the lungs and not enough carbon dioxide is emitted. There are other causes of hypercapnia, as well, including some lung diseases.
Hypercapnia symptoms can range from mild to severe. There are many potential causes of hypercapnia.
This article discusses the symptoms and causes of hypercapnia and outlines some treatment options available to help manage the condition.
In cases where symptoms are mild and develop slowly over time, people may not even realize they have hypercapnia. Therefore, it is important to be aware of both mild and severe symptoms.
The following are considered to be mild symptoms of hypercapnia:
- excessive fatigue
- feeling disoriented
- flushing of the skin
- shortness of breath
These symptoms of hypercapnia may arise from shorter periods of shallow or slow breathing, such as during deep sleep.
They may not always be a cause for concern, as the body is often able to correct the symptoms and balance carbon dioxide levels in the bloodstream without intervention.
However, if the above symptoms persist for several days, it is advisable to see a doctor.
The symptoms of severe hypercapnia require immediate medical attention, as they can cause long-term complications. Some cases may be fatal.
Severe hypercapnia symptoms include:
- depression or paranoia
- hyperventilation or excessive breathing
- irregular heartbeat or arrhythmia
- loss of consciousness
- muscle twitching
- panic attacks
There are many causes of hypercapnia including the following:
Chronic obstructive pulmonary disease or COPD
Chronic bronchitis leads to inflammation and mucus in the airways, while emphysema involves damage to the air sacs or alveoli in the lungs.
Both conditions can cause increased levels of carbon dioxide in the bloodstream.
The main cause of COPD is long-term exposure to lung irritants. According to the National Heart, Lung, and Blood InstituteTrusted Source, cigarette smoke is the most common lung irritant that causes COPD in the United States. Air pollution and exposure to chemicals or dust may also cause COPD.
Although not everyone with COPD will develop hypercapnia, a person’s risk increases as their COPD progresses.
The National Sleep Foundation report that between 5 and 20 percent of adults have sleep apnea.
This common condition is characterized by shallow breathing, or pauses in breathing, during sleep. It can interfere with the level of oxygen in the bloodstream and throw off the body’s balance of carbon dioxide and oxygen.
Sleep apnea symptoms include:
- daytime sleepiness
- headaches upon waking
- difficulty concentrating
Rarely, a genetic condition where the liver fails to produce enough alpha-1-antitrypsin (AAT) can cause hypercapnia. Alpha-1-antitrypsin is a protein that is necessary for lung health, so AAT deficiency is a risk factor for COPD development.
Nerve disorders and muscular problems
In some people, the nerves and muscles necessary for proper lung function may not work correctly. For example, muscular dystrophy can cause the muscles to weaken, eventually leading to breathing problems.
Other disorders of the nervous or muscular systems that can contribute to hypercapnia include:
- Amyotrophic lateral sclerosis (ALS), a progressive disease that affects nerve cells in the brain and spinal cord.
- Encephalitis or when a person has inflammation of the brain.
- Guillain-Barré syndrome that can be caused by an abnormal immune response.
- Myasthenia gravis, a chronic disease that can weaken the skeletal muscles responsible for breathing.
Other causes of high blood levels of carbon dioxide include:
- Activities that impact breathing, including diving or ventilator use.
- Brainstem stroke, which can affect breathing.
- Hypothermia, a medical emergency caused by rapid heat loss from the body.
- Obesity hypoventilation syndromes when overweight people cannot breathe deeply or quickly enough.
- An overdose of certain drugs, such as opioids or benzodiazepines.